Rheumatoid arthritis (RA)
What is RA?
An auto-immune systemic disease. Requires life-long treatment. Individual patients with RA appear to have different pathogenic mechanisms, due to different treatment response. Two major “types”: seropositive and seronegative, with seropositive presenting more severe symptoms.
Risk factors
- First-degree relative with RA
- Gene HLA-DR
- Smoking
- Overweight
- Infections
What are the symptoms?
Typical patient
- Age 30-70
- 70% women
- Symmetrical polyarthritis of hands and feet.
- Common to have morning stiffness lasting more than 30 minutes.
Diagnosis
- Number and location of affected joints
- Duration of disease >= 6 weeks
- Acute-phase reactants
- X-ray for bone erosion
- Serum samples to determine seropositive/seronegative
- Rheumatiod factor (RF)
- Anti-citrullinated protein antibodies (ACPAs)
How can it be treated?
- DMARDs (Disease-Modifying Antirheumatic Drugs)
- Conventional
- Methotrexate (folate antagonist, L04AX03)
- Typical treatment, 33% response rate.
- Leflunomide (L04AK01)
- Sulfasalazine (A07EC01)
- Hydroxychloroquine (P01BA02)
- Methotrexate (folate antagonist, L04AX03)
- Biologic (either alone or combo with conventionals)
- Anti TNF-alpha
- Adalimumab (L04AB04)
- Certolizumab pegol (L04AB05)
- Etanercept (L04AB01)
- Golimumab (L04AB06)
- Infliximab (L04AB02)
- Anti IL6
- Tocilizumab (L04AC07)
- Sarilumab (L04AC14)
- B-cell depletion
- Rituximab (L01FA01)
- T-Cell co-stimulatory inhibitor
- Abatacept (L04AA24)
- Anti TNF-alpha
- Targeted synthetic
- JAK inhibitor
- Tofacitinib (L04AF01)
- Baricitinib (L04AF02)
- Upadacitinib (L04AF03)
- JAK inhibitor
- Conventional
References
- https://www.nejm.org/doi/full/10.1056/NEJMra2103726
- https://www.youtube.com/watch?v=e_NZk8nFSPA